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[3] history of cancer within the past five years

These intermediates induce mitochondrial oxidative stress by covalently binding to mitochondrial constituents including lipoylated dihydrolipoamide S-acetyltransferase (DLAT), leading to its aggregation and triggering regulated cell death via cuproptosis

Compared to urinary PAHs metabolites, PAH-DNA adducts have a longer half-life in vivo, which can comprehensively respond to the exposure, uptake, metabolism, and DNA damage repair of PAHs in the organism 35

Our providers will schedule regular follow-ups to monitor your progress, address any questions, and adjust your care plan as needed to ensure your safety and success