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Vitamin B12 deficiency results in megaloblastic anemia, GI lesions, and neurologic damage (which begins with an inability to produce myelin and is followed by gradual degeneration of the axon and nerve head)
This causes abnormally large cells which reduces the ability of those cells to carry oxygen from enlarged cells in the bone marrow which in turn causes symptoms of anaemia
This ensures absorption in individuals who cannot absorb B12 effectively from food or supplements
Lifelong treatment may be needed when the body is unlikely to regain the ability to absorb enough vitamin B12, such as autoimmune gastritis or the major surgical causes identified by NICE
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