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While the cysteine decomposition is coupled with mitochondrial bioenergetics in the cells with intact KEAP1-NRF2 system, in NRF2-addicted cancer cells, most of the available cysteine appears to flow into the anabolic pathway, namely, glutathione synthesis and leaves the catabolic pathway in mitochondria, which is consistent with the observation that mitochondrial respiration is limited in NRF2-addicted cancer cells (Sayin et al
WynneKParkAJSmallCJMeeranKGhateiMAG.S.Frostet al
Long-acting formulations produce more injection site reactions than short-acting ones
Kt cu v mi hng Sn phm c kt cu lng, trng c, thm kh nhanh trn da
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