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2013;36(Supplement 2):S245-S252

Vanella A, Russo A, Acquaviva R, Campisi A, Di Giacomo C, Sorrenti V, Barcellona ML

In some hypertrophic and failing contexts, inadequate FAO contributes to energetic failure, and pan-ERR agonists that enhance cardiac fatty acid metabolism and mitochondrial function improved heart failure phenotypes ( 3.2 Glucose uptake, glycolysis, pyruvate-lactate flux, and anaplerosis Increased glucose uptake and glycolysis are among the most reproducible features of hypertrophic remodeling, but the fate of glucose carbon varies