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Results Neuronal GCLC knockout leads to brain atrophy accompanied by neuronal cell death To examine how the declines in GCLC and glutathione might affect brain homeostasis, we prepared GCLC floxed X CaMKII-Cre (GCLC-KO) mice whose GCLC expression is deleted in calcium/calmodulin-dependent protein kinase II (CaMKII)-positive neurons

At the end of the study, final blood samples were taken, and the animals were euthanized by cardiac injection

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Exploring the Mechanism of Action The efficacy of dihexa peptide lies in its ability to interact with particular brain receptors that regulate synaptic plasticity