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Further mechanistic investigations determined that the elevation of intracellular GSSG levels resulted in NF-B inhibition by facilitating glutathionylated IKK- (IKK--SSG) formation, leading to enhanced TNF hepatotoxicity

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These results reinforce the idea that IGF- 1 signaling maintains microvascular endothelial function through NO-dependent vasodilation [18]

Lateef OM, Akintubosun MO