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The suppressive actions of insulin on GH release occur independently of IGF-I, since the selective inactivation of the insulin receptor and IGF-I receptor (Insr and IgfIr) genes in mouse somatotrophs does not affect these effects of insulin [156], despite the fact that in the liver, insulin facilitates the expression of IGF-I, which suppresses the synthesis and secretion of GH
Bile acid metabolism, modulated by receptors such as TGR5, FXR, and PXR, further influences lipid homeostasis and vascular health
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