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In contrast, GLP-1RAs are designed to resist degradation by DPP-4, enabling sustained high plasma concentrations and prolonged systemic effects
the reasons explaining this discrepancy are not completely known, but additional discussion of this topic can be found elsewhere
A central challenge is to distinguish true causal propagation from repeated exposure to a shared microenvironmental insult
There is no receptor overlap, no pathway competition, and no known pharmacokinetic interaction between them
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