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Increased Wnt signaling during aging alters muscle stem cell fate and increases fibrosis

As discussed above, a study of the enterogastrone effects of GLP-1 and PYY in dogs, where the GLP-1 receptor antagonist exendin 939 was incapable of blocking the inhibitory actions of GLP-1 (71), suggested that another receptor was involved

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Both are shown in animal models to be particularly adept at suppressing TNF-alpha, a cytokine that is heavily linked with inflammatory bowel disease [16, 17, 18]