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Evidence for NAD(+)-dependent histone dynamics and tunneling associated conformational transitions in circadian deacetylase SIRT1

This small molecule works by selectively inhibiting nicotinamide N-methyltransferase (NNMT), an enzyme that plays a crucial role in cellular energy metabolism and fat storage regulation [1]

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This simultaneous tri-receptor engagement represents a mechanistic departure from earlier compounds in the incretin class, which targeted one or two of these receptors