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23, 24, 25 Despite the reliable and potent inhibitory effects of pharmacological activation of GLP-1R on feeding and body weightparadoxically, pharmacological blockade of central GLP-1R, and especially knockout studies of whole-body or neuronal GLP-1R, produce only minor changes in feeding or body weight

Ca 2+ overload, which is a crucial event in eryptosis, leads to the opening of Ca 2+ -activated K + channel KCa3.1, known as the Gardos channel [90]

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