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While increasing evidence suggests that mitochondrial structure and dynamic regulators are impacted by early postnatal GA exposure, it remains unclear if these changes are a direct result of GA action, or represent downstream responses to other GA-induced damages and processes
Among them, oxidative damage to the autonomic nervous system due to iron accumulation associated with diabetes may be due to changes in the NRF2-GPX4 pathway in ferroptosis (113, 114), the transcription factor that regulates SLC7A11 (51) can effectively prevent oxidative damage in various pathological conditions upon NRF2 activation (115, 116)
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Pregnancy: Insufficient safety data
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