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We find that -arrestin recruitment to GLP-1Rs during sustained agonist exposure has the opposite effect on insulin release to the known positive role of -arrestin-1 during acute GLP-1R stimulation in beta cells 14
One without the other is a compromised system
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Alternatively, a pre-existing small population of open conformations can accommodate the peptide hormone smoothly and this may trigger the transition of GLP-1R from the closed to the otherwise energetically unfavorable open conformation (Fig
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